Definition/General

Introduction:
-Seminoma is a malignant germ cell tumor of the testis
-Represents 50% of testicular germ cell tumors
-Shows uniform cellular morphology
-Derived from primordial germ cells
-Radiosensitive tumor.
Origin:
-Arises from germ cells via germ cell neoplasia in situ (GCNIS)
-Shows blocked differentiation
-Maintains pluripotent characteristics
-Associated with isochromosome 12p.
Classification:
-WHO 2022: Seminoma
-Classic seminoma (most common)
-Seminoma with syncytiotrophoblastic cells
-Anaplastic seminoma (rare)
-Part of germ cell tumor family.
Epidemiology:
-Peak incidence 3rd-4th decades
-Later age than nonseminomatous GCT
-White population predominance
-Cryptorchidism risk factor
-Bilateral in 2-5%.

Clinical Features

Presentation:
-Painless testicular enlargement
-Firm, non-tender mass
-Gradual onset
-Does not transilluminate
-May present with metastases
-Retroperitoneal lymphadenopathy.
Symptoms:
-Testicular swelling
-Heaviness in scrotum
-Dull ache (20%)
-Back pain (retroperitoneal disease)
-Dyspnea (pulmonary metastases)
-Weight loss.
Risk Factors:
-Cryptorchidism (5-10x risk)
-Family history
-Infertility
-Klinefelter syndrome
-Testicular dysgenesis syndrome
-Previous germ cell tumor.
Screening:
-Testicular self-examination
-Clinical examination
-Scrotal ultrasound
-Tumor markers (LDH, βhCG)
-CT imaging for staging.

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Gross Description

Appearance:
-Well-demarcated, lobulated mass
-Homogeneous tan-gray cut surface
-Soft consistency
-Replaces testicular parenchyma
-Minimal necrosis
-Fibrous septa.
Characteristics:
-Fleshy appearance
-Fish-flesh consistency
-Uniform coloration
-Well-circumscribed
-Lobulated pattern
-Granular surface.
Size Location:
-Variable size 2-10 cm
-Usually unilateral
-Replaces normal testis
-May involve epididymis
-Spermatic cord extension
-Scrotal wall invasion.
Multifocality:
-Usually unifocal
-Bilateral synchronous (2%)
-Bilateral metachronous (1-3%)
-Associated GCNIS in adjacent testis
-Higher bilateral risk with cryptorchidism.

Microscopic Description

Histological Features:
-Uniform large cells
-Clear to eosinophilic cytoplasm
-Distinct cell borders
-Large nuclei with prominent nucleoli
-Lymphocytic infiltrate
-Granulomatous reaction.
Cellular Characteristics:
-Large polygonal cells
-Abundant clear cytoplasm
-Glycogen-rich
-Well-defined cell membranes
-Large vesicular nuclei
-1-2 prominent nucleoli.
Architectural Patterns:
-Sheets and lobules
-Fibrous septa with lymphocytes
-Granulomatous inflammation
-Syncytiotrophoblastic giant cells (some cases)
-No glandular formation
-Solid growth.
Grading Criteria:
-No formal grading
-Anaplastic seminoma (high mitotic rate, pleomorphism)
-Presence of GCNIS
-Lymphovascular invasion
-Rete testis invasion.

Immunohistochemistry

Positive Markers:
-OCT3/4 - positive (nuclear, sensitive/specific)
-D2-40 (PODOPLANIN) - positive
-CD117 - positive
-Placental alkaline phosphatase - positive
-SALL4 - positive
-SOX17 - positive.
Negative Markers:
-CD30 - negative (vs embryonal carcinoma)
-Cytokeratin - negative
-EMA - negative
-S-100 - negative
-Vimentin - negative
-TTF1 - negative.
Diagnostic Utility:
-OCT3/4 most useful (nuclear staining)
-D2-40 highly sensitive
-CD117 supports diagnosis
-Negative CD30 excludes embryonal carcinoma
-SALL4 germ cell marker
-SOX17 specific for seminoma.
Molecular Subtypes:
-Classic seminoma: OCT3/4+, D2-40+, CD117+
-With syncytiotrophoblastic cells: βhCG+
-All seminomas: SALL4+, SOX17+
-Anaplastic: Same markers, high proliferation.

Molecular/Genetic

Genetic Mutations:
-Isochromosome 12p (i(12p)) - pathognomonic
-12p amplification
-KIT mutations (rare)
-KRAS mutations
-PIK3CA mutations
-Aneuploidy common.
Molecular Markers:
-Chromosome 12p gain
-KIT/PDGFRA pathway
-PI3K/AKT pathway
-p53 wild-type
-DNA hypomethylation
-MicroRNA-371/372/373 cluster.
Prognostic Significance:
-Stage most important
-Tumor size >4 cm: adverse
-Rete testis invasion: intermediate risk
-Lymphovascular invasion: adverse
-Age >40: intermediate risk
-Anaplastic: rare, poor prognosis.
Therapeutic Targets:
-Radiation therapy (radiosensitive)
-Platinum-based chemotherapy (BEP, EP)
-Surveillance (stage I)
-Immunotherapy (refractory)
-High cure rates (>95%).

Differential Diagnosis

Similar Entities:
-Embryonal carcinoma
-Large cell lymphoma
-Metastatic carcinoma
-Leydig cell tumor
-Spermatocytic tumor
-Solid variant yolk sac tumor.
Distinguishing Features:
-Seminoma: OCT3/4+, CD30-, lymphocytic infiltrate
-Embryonal carcinoma: CD30+, necrosis, glandular pattern
-Lymphoma: CD45+
-Carcinoma: Organ-specific markers
-Leydig cell: Inhibin+
-Spermatocytic: Older age, OCT3/4-.
Diagnostic Challenges:
-Seminoma vs embryonal carcinoma
-Burned-out seminoma
-Seminoma with extensive necrosis
-Anaplastic seminoma
-Mixed germ cell tumors.
Rare Variants:
-Seminoma with syncytiotrophoblastic cells
-Anaplastic seminoma
-Microcystic seminoma
-Tubular seminoma.

Sample Pathology Report

Template Format

Sample Pathology Report

Complete Report: This is an example of how the final pathology report should be structured for this condition.

Specimen Information

Radical orchiectomy specimen, testis measuring [X] cm

Tumor Description

Seminoma measuring [X] cm with tan-gray homogeneous cut surface

Microscopic Features

Uniform large cells with clear cytoplasm, distinct borders, and lymphocytic infiltrate. [Syncytiotrophoblastic cells present/absent]

GCNIS

Germ cell neoplasia in situ: [present/absent] in adjacent testicular parenchyma

pT Stage

pT[stage] - [staging details]

Lymphovascular Invasion

Lymphovascular invasion: [present/absent]

Immunohistochemistry

OCT3/4: Positive (nuclear), D2-40: Positive, CD30: Negative, CD117: Positive

Final Diagnosis

Seminoma, pT[stage], [size] cm, [risk stratification]