Overview

Definition:
-Biologic therapies for asthma are targeted treatments that modulate specific components of the inflammatory cascade
-Anti-immunoglobulin E (anti-IgE) therapy, exemplified by omalizumab, targets IgE, a key mediator in allergic asthma
-Anti-interleukin-5 (anti-IL-5) therapies, including mepolizumab, reslizumab, and benralizumab, target IL-5 or its receptor, which is crucial for eosinophil proliferation, maturation, and activation, particularly in eosinophilic asthma phenotypes.
Epidemiology:
-Severe asthma affects approximately 5-10% of all asthma patients, with a significant proportion being children
-In pediatrics, allergic asthma is common, often driven by IgE
-Eosinophilic asthma, characterized by elevated blood eosinophil counts, is a distinct phenotype that responds well to anti-IL-5 therapies.
Clinical Significance:
-These biologics offer a crucial therapeutic option for children with severe, uncontrolled asthma despite optimal standard care
-They can reduce exacerbation rates, improve lung function, decrease oral corticosteroid dependence, and enhance quality of life
-Understanding their mechanisms, indications, and adverse effects is vital for pediatricians and trainees preparing for DNB and NEET SS examinations.

Anti Ige Therapy

Mechanism Of Action:
-Omalizumab is a recombinant humanized monoclonal antibody that binds to the Fc region of free IgE, preventing it from binding to mast cells and basophils
-This reduces the release of inflammatory mediators upon allergen exposure.
Indications:
-Approved for children aged 6 years and older with moderate to severe persistent allergic asthma that is inadequately controlled by inhaled corticosteroids (ICS) and long-acting beta-agonists (LABA)
-Requires evidence of sensitization to perennial aeroallergens and elevated IgE levels
-Also indicated for chronic spontaneous urticaria in specific age groups.
Dosing Administration:
-Dosage is based on body weight and serum IgE levels, typically administered every 2 or 4 weeks via subcutaneous injection
-Dosing nomogram guides precise administration
-For asthma, doses range from 75 mg to 375 mg every 2-4 weeks.
Adverse Effects:
-Common side effects include injection site reactions (pain, redness, itching), headache, and pharyngitis
-Rare but serious adverse events include anaphylaxis (risk during initial doses), and a potential increased risk of parasitic infections, especially in endemic areas
-It is crucial to monitor for anaphylaxis closely during administration and for a period afterward.

Anti Il 5 Therapy

Mechanism Of Action:
-Mepolizumab and reslizumab are monoclonal antibodies targeting IL-5, a cytokine essential for eosinophil development and function
-Benralizumab is a monoclonal antibody that binds to the IL-5 receptor alpha subunit on eosinophils, leading to their depletion via antibody-dependent cell-mediated cytotoxicity
-Targeting IL-5 or its receptor reduces eosinophil counts in the blood and airways.
Indications:
-Indicated for children aged 12 years and older (reslizumab) or 6 years and older (mepolizumab, benralizumab) with severe eosinophilic asthma that is not controlled by high-dose ICS and LABA
-Requires demonstration of elevated blood eosinophil counts (e.g., >150 cells/µL at baseline or >300 cells/µL in the past year) and evidence of at least one exacerbation in the past year.
Dosing Administration:
-Mepolizumab: 100 mg subcutaneous injection every 4 weeks, or 300 mg intravenous infusion every 4 weeks for severe asthma
-Reslizumab: 3 mg/kg intravenous infusion every 4 weeks
-Benralizumab: 30 mg subcutaneous injection every 4 weeks for the first 3 doses, then every 8 weeks thereafter.
Adverse Effects:
-Common side effects include nasopharyngitis, headache, and injection site reactions
-Hypersensitivity reactions are possible
-A key consideration is the potential for paradoxical bronchospasm and the risk of eosinophilic granulomatosis with polyangiitis (EGPA) in patients with pre-existing EGPA or other eosinophilic conditions when oral corticosteroids are reduced
-Patients on chronic oral corticosteroids should have their dosage reduced gradually under medical supervision.

Selection And Monitoring

Patient Selection:
-Careful phenotyping is essential
-Anti-IgE is primarily for allergic asthma with elevated IgE and sensitization
-Anti-IL-5 agents are for eosinophilic asthma phenotypes, identified by blood eosinophil counts and often a history of steroid dependence or frequent exacerbations
-Shared decision-making with parents and the child is crucial.
Monitoring Efficacy:
-Efficacy is assessed by reduction in asthma exacerbations, improvement in asthma control scores (e.g., ACT, c-ACT), lung function (FEV1), and reduction in oral corticosteroid use
-Blood eosinophil counts should be monitored, especially when titrating ICS or oral steroids.
Monitoring Safety:
-Regular follow-up is essential to monitor for adverse events, including hypersensitivity reactions, anaphylaxis (for omalizumab), and changes in symptoms suggestive of EGPA (for anti-IL-5 agents)
-Periodic assessment of adherence and technique is also important.

Key Points

Exam Focus:
-Understand the distinct mechanisms of anti-IgE and anti-IL-5 therapies
-Recognize the specific phenotypes (allergic vs
-eosinophilic asthma) and their corresponding biologic treatments
-Memorize the age indications, dosing frequencies, and key adverse events for each biologic
-Differentiate between IL-5 inhibitors and IL-5 receptor blockers.
Clinical Pearls:
-Always confirm IgE levels and allergen sensitization before initiating omalizumab
-For anti-IL-5 therapies, establish baseline eosinophil counts and monitor them during treatment
-Titrate oral corticosteroids cautiously when initiating biologics
-Educate patients and caregivers about the potential for anaphylaxis with omalizumab and EGPA with anti-IL-5 agents.
Common Mistakes:
-Incorrectly selecting a biologic for the wrong asthma phenotype
-Failing to adequately monitor for serious adverse events like anaphylaxis or EGPA
-Not educating patients on proper injection technique or symptom recognition
-Inadequate follow-up to assess treatment response and safety.