Overview/Definition

Definition:
-• Congenital hypothyroidism (CH) is deficiency of thyroid hormone from birth due to thyroid dysgenesis (80-85%), dyshormonogenesis (10-15%), or central hypothyroidism (5%)
-Most common preventable cause of intellectual disability with excellent prognosis if diagnosed and treated early within first 2-4 weeks of life.
Epidemiology:
-• Global incidence 1:2000-4000 newborns with higher rates in certain populations
-In India, estimated incidence 1:2500-3500 newborns based on limited screening data
-Female to male ratio 2:1
-Higher incidence in Down syndrome, preterm infants
-Iodine deficiency affects regional prevalence patterns.
Age Distribution:
-• Neonatal period: Critical window for diagnosis (0-28 days) to prevent irreversible brain damage
-Infancy (1-24 months): Continued high sensitivity to thyroid hormone deficiency for brain development
-Childhood (2-12 years): Growth and developmental impacts
-Adolescence: Pubertal development and final height achievement.
Clinical Significance:
-• High-yield topic for DNB Pediatrics and NEET SS examinations focusing on screening protocols, diagnostic criteria, treatment initiation, and long-term monitoring
-Understanding newborn screening programs, false positive/negative rates, and emergency management essential
-Critical for preventing intellectual disability.

Age-Specific Considerations

Newborn:
-• Neonates (0-28 days): Newborn screening typically performed 48-72 hours after birth when TSH surge occurs
-Preterm infants may need repeat screening due to immature hypothalamic-pituitary-thyroid axis
-Screen before blood transfusion or discharge
-Emergency treatment for severe hypothyroidism with bradycardia, hypothermia.
Infant:
-• Infants (1-24 months): Most critical period for treatment to prevent intellectual disability
-Rapid brain growth requires adequate thyroid hormone
-Monitor growth velocity, developmental milestones closely
-Adjust dosing frequently based on growth
-Higher per-weight levothyroxine requirements compared to older children.
Child:
-• Children (2-12 years): Continue treatment with regular monitoring
-Growth rate excellent indicator of adequacy
-School performance and cognitive development assessment important
-Dosing adjustments less frequent but still weight-based
-Transition from liquid to tablet formulations possible.
Adolescent:
-• Adolescents (12-18 years): Pubertal development requires adequate thyroid hormone
-Final height achievement depends on treatment adequacy
-Medication compliance issues common
-Adult-like dosing approaches
-Transition planning to adult endocrinology care.

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Clinical Presentation

Symptoms:
-• Early neonatal symptoms often subtle: Prolonged jaundice >2 weeks, feeding difficulties, lethargy, constipation
-Later symptoms if untreated: Developmental delays, growth retardation, intellectual disability
-Older children: Fatigue, cold intolerance, weight gain, school performance decline
-Pubertal delays in adolescents.
Physical Signs:
-• Neonatal signs: Macroglossia, umbilical hernia, hypotonia, mottled skin
-Growth parameters: Length/height below percentiles, delayed weight gain
-Facial features: Puffy face, thick lips, enlarged fontanelles
-Cardiovascular: Bradycardia, murmurs
-Developmental: Delayed milestones, hyporeflexia.
Severity Assessment:
-• Mild CH: TSH 20-50 mIU/L, minimal symptoms, normal T4
-Moderate CH: TSH 50-100 mIU/L, some symptoms, low-normal T4
-Severe CH: TSH >100 mIU/L, significant symptoms, low T4
-Central CH: Low/normal TSH with low T4, often part of multiple pituitary hormone deficiency.
Differential Diagnosis:
-• Transient hypothyroidism: Maternal antithyroid drugs, iodine excess/deficiency, prematurity
-Other causes of developmental delay: Genetic syndromes, metabolic disorders
-Growth failure: Growth hormone deficiency, malnutrition, chronic illness
-Neonatal jaundice: Biliary atresia, sepsis, hemolysis.

Diagnostic Approach

History Taking:
-• Family history: Thyroid disorders, autoimmune diseases, consanguinity
-Maternal history: Thyroid disease, medications (antithyroid drugs, iodine), radiation exposure
-Neonatal history: Birth weight, gestational age, jaundice, feeding problems
-Developmental history: Milestone achievements, growth patterns.
Investigations:
-• Newborn screening: TSH measurement from dried blood spot (Guthrie card)
-Confirmatory tests: Serum TSH, free T4, total T4
-Additional tests: Thyroglobulin, thyroid peroxidase antibodies, thyroid ultrasound
-Imaging: Thyroid scintigraphy to differentiate agenesis, dysgenesis, ectopia.
Normal Values:
-• Newborn TSH: <20 mIU/L (screening), <10 mIU/L (serum)
-Free T4: 10-25 pmol/L (0.8-2.0 ng/dL)
-Total T4: 65-175 nmol/L (5-13.5 μg/dL)
-Target ranges on treatment: TSH 0.5-2.0 mIU/L (first year), 0.5-4.0 mIU/L (after 1 year)
-Free T4 upper half of normal range.
Interpretation:
-• Screening positive: TSH >20 mIU/L requires immediate confirmatory testing
-Confirmatory: TSH >10 mIU/L with low/low-normal T4 confirms diagnosis
-Transient forms: Normalize by 3-6 months, may require temporary treatment
-Central CH: Low T4 with inappropriately low/normal TSH.

Management/Treatment

Acute Management:
-• Emergency situations: Severe hypothyroidism with cardiovascular compromise requires immediate IV levothyroxine
-Newborn diagnosis: Start treatment immediately, do not wait for confirmatory results if high clinical suspicion
-Initial dosing: 10-15 μg/kg/day levothyroxine orally.
Chronic Management:
-• Long-term levothyroxine replacement therapy for life
-Regular monitoring: TSH, free T4 every 2-4 weeks initially, then every 2-3 months first year, every 3-4 months thereafter
-Dose adjustments: Based on TSH, free T4, growth velocity, development
-Brand consistency important.
Lifestyle Modifications:
-• Medication timing: Give on empty stomach, 30-60 minutes before feeding
-Avoid simultaneous administration with iron, calcium, soy formula
-Regular meal timing to optimize absorption
-Normal physical activity encouraged
-Adequate iodine intake in diet.
Follow Up:
-• Intensive monitoring first year: Every 2-4 weeks initially, then every 2-3 months
-Childhood monitoring: Every 3-4 months with growth and development assessment
-Adolescence: Every 4-6 months with pubertal assessment
-Adult transition: Gradual transfer to adult endocrinology.

Age-Specific Dosing

Medications:
-• Levothyroxine starting doses: Neonates (0-3 months): 10-15 μg/kg/day
-Infants (3-6 months): 8-10 μg/kg/day
-Children (6 months-2 years): 6-8 μg/kg/day
-Children (2-12 years): 4-6 μg/kg/day
-Adolescents (12-18 years): 2-4 μg/kg/day, transitioning to adult dosing.
Formulations:
-• Available strengths: 25, 50, 75, 88, 100, 112, 125, 137, 150, 175, 200 μg tablets
-Liquid formulation available for infants and children with swallowing difficulties
-Crushing tablets: Can be crushed and mixed with small amount of water, breast milk, or formula (not soy-based).
Safety Considerations:
-• Drug interactions: Iron supplements, calcium, soy products reduce absorption (separate by 4 hours)
-Overtreatment risks: Tachycardia, irritability, sleep disturbances, accelerated bone maturation
-Undertreatment: Growth failure, developmental delays, intellectual disability.
Monitoring:
-• Growth parameters: Weight, length/height, head circumference plotted on growth charts
-Developmental assessment: Milestone achievements, cognitive testing
-Laboratory monitoring: TSH, free T4 levels
-Cardiac assessment: Heart rate, blood pressure
-Bone age assessment if growth concerns.

Prevention & Follow-up

Prevention Strategies:
-• Primary prevention: Adequate maternal iodine intake during pregnancy
-Universal newborn screening programs for early detection
-Public health measures: Iodized salt programs, population iodine supplementation
-Genetic counseling for families with hereditary forms.
Vaccination Considerations:
-• No specific vaccine modifications required
-Follow routine immunization schedule
-Thyroid hormone replacement does not affect vaccine efficacy or safety
-Live vaccines can be given normally
-Monitor for intercurrent illnesses affecting thyroid hormone requirements.
Follow Up Schedule:
-• First year: Every 2-4 weeks until stable, then every 2-3 months
-Childhood: Every 3-4 months with interim visits for dose adjustments
-Adolescence: Every 4-6 months
-Sick visits: During illnesses that may affect absorption or metabolism
-Pre-conception counseling for females.
Monitoring Parameters:
-• Laboratory: TSH, free T4 levels targeting normal ranges
-Growth: Linear growth velocity, weight gain patterns
-Development: Milestone achievements, IQ testing at school age
-Compliance: Medication adherence assessment
-Quality of life: School performance, social functioning.

Complications

Acute Complications:
-• Overtreatment: Hyperthyroidism symptoms (tachycardia, irritability, sleep disturbances), accelerated bone maturation, craniosynostosis in infants
-Undertreatment: Developmental delays, intellectual disability, growth failure
-Medication errors: Wrong dosing, formulation issues.
Chronic Complications:
-• Intellectual disability: If treatment delayed beyond 2-4 weeks or inadequately treated
-Growth failure: Final height compromise with late or inadequate treatment
-Learning difficulties: Subtle cognitive deficits even with early treatment
-Behavioral problems: ADHD-like symptoms, social difficulties.
Warning Signs:
-• Signs of overtreatment: Rapid heart rate, irritability, excessive activity, sleep problems
-Signs of undertreatment: Lethargy, poor growth, developmental delays, persistent symptoms
-Medication absorption issues: Vomiting, diarrhea, drug interactions.
Emergency Referral:
-• Immediate pediatric endocrinology referral for: Diagnostic uncertainty, treatment resistance, severe hypothyroidism
-Cardiology referral for cardiac complications
-Developmental assessment for delays
-Genetics referral for familial forms or associated syndromes.

Parent Education Points

Counseling Points:
-• Congenital hypothyroidism is treatable condition with excellent prognosis if treated early and adequately
-Lifelong medication required but allows normal development
-Importance of medication compliance and regular monitoring
-Early treatment prevents intellectual disability.
Home Care:
-• Medication administration: Give on empty stomach, same time daily, avoid mixing with soy, iron, calcium
-Store medication in cool, dry place
-Monitor for signs of over/under treatment
-Maintain medication supply, refill prescriptions timely
-Keep medication diary.
Medication Administration:
-• Levothyroxine timing: 30-60 minutes before first feeding
-Crushing tablets: Mix with small amount of water, give immediately
-Avoid grapefruit juice, high-fiber foods near medication time
-Consistency in timing and administration method important for stable levels.
When To Seek Help:
-• Contact healthcare provider for: Signs of overtreatment (rapid heartbeat, irritability), undertreatment (lethargy, poor growth), missed doses for several days
-Illness affecting medication absorption
-Concerns about growth or development
-Medication supply issues or side effects.