Overview

Definition: Postoperative delirium (POD) is an acute, fluctuating disturbance of consciousness characterized by inattention, disorganized thinking, and altered level of consciousness, occurring in the perioperative period.
Epidemiology:
-Incidence varies widely based on patient population and surgical procedure, ranging from 10% in younger patients undergoing minor surgery to over 50% in elderly patients undergoing major elective or emergency surgery
-Higher rates seen in cardiac, orthopedic, and abdominal surgeries.
Clinical Significance:
-POD is associated with prolonged hospital stay, increased morbidity and mortality, higher rates of institutionalization, and long-term cognitive decline
-Early recognition and management are crucial to mitigate these adverse outcomes.

Risk Factors

Patient Factors:
-Advanced age (>65 years)
-Pre-existing cognitive impairment or dementia
-Delirium in the past
-Low socioeconomic status
-Poor baseline functional status
-Comorbidities (e.g., hypertension, diabetes, heart failure, renal impairment)
-Sensory deficits (hearing, vision)
-Alcohol or substance abuse.
Surgical Factors:
-Emergency surgery
-Major surgery
-Prolonged operative time
-Intraoperative blood loss
-Use of general anesthesia (vs
-regional)
-Specific anesthetic agents.
Postoperative Factors:
-Pain
-Sleep deprivation
-Immobility
-Fluid and electrolyte imbalances
-Hypoxia
-Hypoglycemia
-Infection
-Polypharmacy
-Urinary catheterization
-Physical restraints
-Intensive care unit (ICU) environment.

Clinical Presentation

Symptoms:
-Acute onset of fluctuating mental status
-Inattention and distractibility
-Disorganized thinking or speech
-Altered level of consciousness (hyperactive, hypoactive, or mixed)
-Hallucinations (visual, auditory)
-Delusions
-Agitation or withdrawal
-Disorientation (time, place, person)
-Sleep-wake cycle disturbances.
Signs:
-Inability to focus or maintain attention
-Random, incoherent speech
-Fluctuating vital signs (tachycardia, hypertension, or hypotension)
-Evidence of distress or anxiety
-Psychomotor agitation or retardation
-May appear drowsy or stuporous.
Diagnostic Criteria:
-Based on DSM-5 criteria for delirium: 1
-Disturbance of attention and awareness
-2
-Acute onset and fluctuating course
-3
-Disturbance in cognition (memory deficit, disorientation, language, visual-spatial ability, perception)
-4
-Not better explained by another neurocognitive disorder
-5
-Not due to substance intoxication/withdrawal or another medical condition
-The Confusion Assessment Method (CAM) is a widely used screening tool.

Prevention Strategies

Preoperative Optimization:
-Identify and address risk factors
-Optimize comorbidities
-Cognitive assessment
-Patient and family education about POD
-Medication review to discontinue high-risk drugs.
Intraoperative Measures:
-Minimize anesthetic agents
-Maintain hemodynamic stability
-Ensure adequate oxygenation and ventilation
-Minimize blood loss
-Use of intraoperative monitoring
-Consider regional anesthesia where appropriate.
Postoperative Care Bundle:
-Early mobilization
-Early removal of catheters
-Pain management without over-sedation
-Sensory aids (glasses, hearing aids)
-Sleep hygiene measures
-Hydration and nutrition
-Prevention of infection
-Avoidance of physical restraints
-Regular reorientation and reassurance
-Use of low-risk medications.

Management

Initial Assessment:
-Rapid assessment of airway, breathing, and circulation
-Assess for reversible causes of delirium (e.g., hypoxia, hypoglycemia, electrolyte disturbances, infection, drug toxicity, withdrawal)
-Rule out other acute neurological events (stroke, seizure).
Addressing Precipitants:
-Treat underlying causes aggressively
-Manage pain effectively with multimodal analgesia
-Optimize fluid and electrolyte balance
-Treat infections promptly
-Correct hypoxia
-Manage hypoglycemia
-Discontinue or adjust offending medications.
Pharmacological Management:
-Antipsychotics (e.g., haloperidol, olanzapine, quetiapine) are generally reserved for severe agitation or psychosis that poses a risk to the patient or staff
-Use the lowest effective dose for the shortest duration
-Avoid benzodiazepines in most cases, as they can worsen delirium (except in alcohol or benzodiazepine withdrawal).
Non Pharmacological Management:
-Environmental modifications: quiet room, good lighting, familiar objects
-Frequent reorientation and reassurance by staff
-Family involvement and support
-Promote sleep-wake cycles
-Encourage early ambulation and physical therapy
-Sensory stimulation
-Avoid excessive noise and stimulation
-Gradual reduction of monitoring equipment.

Complications

Associated Morbidity:
-Increased risk of falls
-Poor wound healing
-Prolonged mechanical ventilation
-Higher rates of deep vein thrombosis (DVT) and pulmonary embolism (PE)
-Urinary tract infections (UTIs)
-Pneumonia
-Pressure ulcers
-Longer length of stay
-Higher readmission rates.
Long Term Sequelae:
-Persistent cognitive impairment
-Increased risk of developing dementia
-Functional decline and loss of independence
-Increased mortality
-Negative impact on quality of life.
Prevention Strategies:
-Adherence to comprehensive delirium prevention protocols
-Multidisciplinary team approach involving physicians, nurses, therapists, and pharmacists
-Patient and family education
-Continuous quality improvement initiatives.

Key Points

Exam Focus:
-Recognize POD as a common and serious complication
-Know the risk factors (patient, surgical, postoperative)
-Understand the diagnostic criteria (CAM)
-Prioritize prevention strategies with the "ABCDE" bundle or similar protocols
-Manage underlying causes first
-judicious use of antipsychotics for agitation only.
Clinical Pearls:
-The hypoactive subtype of delirium is often missed but carries a poor prognosis
-Treat pain aggressively but monitor for over-sedation
-Non-pharmacological measures are the cornerstone of management
-Early mobilization is key
-Consider delirium as a sign of underlying physiological distress.
Common Mistakes:
-Attributing all confusion in elderly patients to dementia without considering acute causes
-Over-reliance on antipsychotics without addressing underlying precipitants
-Underestimating the importance of non-pharmacological interventions
-Failure to implement comprehensive prevention protocols.