Overview

Definition:
-Zone II of the flexor tendon system in the hand, often termed "no man's land", extends from the distal palmar crease to the insertion of the flexor digitorum superficialis
-Injuries in this zone are complex due to the tight space, lack of mesotenon, and the presence of vital structures, making tendon repair and subsequent rehabilitation critically important for functional recovery.
Epidemiology:
-Flexor tendon injuries are common, particularly in younger, active individuals
-Zone II injuries constitute a significant proportion of these
-Factors like type of injury (laceration, avulsion), mechanism, and patient compliance influence outcomes.
Clinical Significance:
-Successful repair and meticulous rehabilitation in Zone II are paramount to restore full digital motion and prevent long-term disability
-Adhesions are a major concern, leading to restricted motion and functional impairment, directly impacting a patient's quality of life and ability to perform daily activities and work
-Early and coordinated management is key.

Clinical Presentation

Symptoms:
-Inability to actively flex the injured finger at the distal interphalangeal (DIP) and proximal interphalangeal (PIP) joints
-Pain at the site of injury
-Palpable defect or gap if the tendon is completely severed
-Bleeding may be present depending on the wound
-Loss of passive extension of the PIP joint can suggest a "buttonhole" deformity if the flexor digitorum profundus (FDP) is retracted proximally.
Signs:
-Loss of active flexion at the PIP joint and DIP joint
-Inability to make a fist
-Passive flexion may be possible but limited
-Tenderness over the palmar aspect of the finger
-Visible wound or scar
-A positive "lag" of the finger when attempting to flex the adjacent fingers (indicating FDP rupture).
Diagnostic Criteria:
-Diagnosis is primarily clinical, based on the mechanism of injury, patient history, and physical examination findings of loss of active flexion
-If the wound is complex or the diagnosis is unclear, imaging may be considered, though direct visualization during exploration is definitive.

Diagnostic Approach

History Taking:
-Detailed history of the injury mechanism (e.g., sharp laceration, crush injury, avulsion)
-Time since injury
-Patient's occupation and dominant hand
-Previous hand surgeries or injuries
-Assessment of tetanus immunization status
-Evaluation of sensation and vascular status distal to the injury.
Physical Examination:
-Careful assessment of all digits for tendon injuries
-Assess active and passive range of motion of all finger joints (MCP, PIP, DIP)
-Assess sensation (light touch, two-point discrimination)
-Assess vascularity (capillary refill, pulse)
-Evaluate for concomitant nerve or vessel injury.
Investigations:
-Radiographs are primarily used to rule out bony injury (fractures, dislocations) or foreign bodies
-Ultrasound may sometimes be used to assess tendon integrity and extent of injury preoperatively, but it is not routinely necessary
-The definitive diagnosis and assessment of tendon status are made during surgical exploration.
Differential Diagnosis:
-Extensor tendon injury
-Nerve injury (digital nerve laceration)
-Vascular injury
-Joint capsule injury
-Contusion or sprain
-Fracture-dislocation
-Skin and soft tissue loss.

Management

Initial Management:
-Thorough wound irrigation and debridement
-Assess for tendon, nerve, and vascular integrity
-If tendon is injured, ensure adequate visualization
-Gentle splinting to protect the repaired tendon, typically in a position of slight flexion
-Tetanus prophylaxis if indicated
-Antibiotic prophylaxis is often administered
-Urgent surgical consultation is required.
Surgical Management:
-Surgical repair is indicated for complete flexor tendon injuries in Zone II
-Primary repair aims to approximate tendon ends with minimal tension
-Techniques include modified Kessler, Savage, or epitendinous suture techniques using fine, non-absorbable monofilament sutures (e.g., 4-0, 5-0, 6-0 Prolene or Nylon)
-Avoidance of excessive knots or bulky suture material within the tendon sheath is crucial to minimize adhesion formation
-Repair should ideally be done within 7-10 days of injury, though delayed repairs are possible
-Consideration for tendon grafting if the gap is too large for primary repair or if there is significant tendon loss
-Nerve repair should be performed concurrently if digital nerves are severed
-Consideration for vascular repair if indicated.
Rehabilitation Protocol:
-Postoperative rehabilitation is critical and typically involves a structured protocol
-It starts with immobilisation in a protective splint (e.g., volar splint or dynamic splint)
-Passive range of motion exercises are initiated early (often within 3-5 days), followed by controlled active motion and tendon gliding exercises
-A common protocol includes: 1
-Early passive flexion and extension (within splint limitations)
-2
-Tendon gliding exercises (e.g., hook fist, straight fist, full fist)
-3
-Gentle active flexion exercises
-4
-Progressive strengthening
-This phased approach aims to prevent adhesions while allowing tendon healing
-Supervision by a trained hand therapist is essential.
Supportive Care:
-Pain management with analgesics
-Edema control with elevation and compression
-Wound care and monitoring for signs of infection
-Patient education on splint wear, exercise regimen, and danger signs.

Complications

Early Complications:
-Tendon rupture during rehabilitation
-Infection
-Wound dehiscence
-Stiffness due to premature adhesion formation
-Extensor lag due to inadvertent injury to extensor mechanism
-Nerve injury during repair
-Volar plate subluxation.
Late Complications:
-Persistent stiffness
-Adhesions causing restricted motion
-Triggering of the digit
-Tendon bowstringing
-Malunion or non-union of bony fragments if associated fracture
-Chronic pain
-Loss of grip strength.
Prevention Strategies:
-Meticulous surgical technique to minimize trauma and knotting
-Early and diligent adherence to a structured rehabilitation protocol
-Close supervision by a hand therapist
-Appropriate splinting
-Patient compliance with exercises and activity restrictions
-Prophylactic antibiotics and tetanus immunization.

Prognosis

Factors Affecting Prognosis:
-Zone of injury (Zone II is most challenging)
-Type and extent of injury
-Quality of tendon repair
-Patient age and health status
-Strict adherence to postoperative rehabilitation protocol
-Presence of concomitant nerve or vessel injury
-Surgeon's experience
-Avoidance of infection.
Outcomes:
-With optimal repair and rehabilitation, good to excellent functional outcomes can be achieved, including full or near-full range of motion
-However, some degree of stiffness or limitation may persist, especially with challenging injuries or poor compliance
-Full return to pre-injury level of function, particularly for manual laborers, may take several months
-Poor outcomes are characterized by significant stiffness, limited motion, and chronic pain.
Follow Up:
-Regular follow-up with the hand surgeon and hand therapist is crucial
-Typically, initial follow-up is at 1-2 weeks post-op for suture removal and assessment of wound healing
-Subsequent visits monitor progress of rehabilitation, range of motion, and to adjust the protocol as needed
-Long-term follow-up may be required for several months to ensure sustained functional recovery and address any emerging issues.

Key Points

Exam Focus:
-Zone II is "no man's land" due to high adhesion risk
-Meticulous surgical repair and early, structured rehabilitation are key
-The goal is to achieve tendon healing without excessive scarring
-Tendon gliding exercises are fundamental
-Know the different phases of rehabilitation and common complications like stiffness and rupture.
Clinical Pearls:
-Use fine monofilament non-absorbable sutures for repair
-Avoid bulky knots and excessive tension
-A circumferential epitendinous suture provides optimal strength
-Early passive motion is vital to prevent adhesions
-If passive motion is possible but active is not, suspect tendon injury
-Concurrent digital nerve repair is often necessary.
Common Mistakes:
-Aggressive early active motion leading to rupture
-Insufficient or delayed rehabilitation leading to adhesions and stiffness
-Inadequate surgical repair technique causing gapping or instability
-Ignoring concomitant neurovascular injuries
-Poor patient education regarding the importance of rehabilitation compliance.