Overview

Definition:
-Superior Mesenteric Artery (SMA) injury refers to damage to the main artery supplying blood to the small intestine, most of the large intestine, and parts of the pancreas
-This can result from blunt or penetrating trauma, leading to bowel ischemia, hemorrhage, and potentially fatal outcomes if not promptly managed.
Epidemiology:
-SMA injuries are relatively uncommon, accounting for a small percentage of all visceral artery injuries
-They are more frequently seen in penetrating trauma (stab wounds, gunshot wounds) than blunt trauma
-High-energy blunt trauma, such as from motor vehicle accidents, can cause shear forces leading to intimal tears or even complete transection.
Clinical Significance:
-SMA injuries are a surgical emergency due to the critical role of the SMA in bowel perfusion
-Inadequate blood supply leads to mesenteric ischemia, which can rapidly progress to bowel infarction, sepsis, multiorgan failure, and death
-Prompt diagnosis and definitive management are crucial for patient survival and preservation of bowel viability.

Clinical Presentation

Symptoms:
-Severe abdominal pain, often out of proportion to physical findings
-Nausea and vomiting
-Hematemesis or melena (if injury is proximal or associated with duodenal injury)
-Abdominal distension
-Signs of hypovolemic shock: tachycardia, hypotension, pallor, altered mental status
-Absence of palpable pulses distal to the injury (rare).
Signs:
-Tenderness and guarding of the abdomen
-Rebound tenderness
-Decreased bowel sounds or absent bowel sounds
-Abdominal distension
-Pulsatile abdominal mass (if pseudoaneurysm)
-Hemorrhagic shock
-Signs of peritonitis
-Evidence of external trauma (lacerations, contusions, seatbelt marks).
Diagnostic Criteria:
-No specific diagnostic criteria exist
-diagnosis is based on high clinical suspicion in the setting of abdominal trauma and confirmed by imaging
-The American Association for the Surgery of Trauma (AAST) organ injury scale (OIS) for the small bowel and mesentery can be used for grading severity of associated injuries, which often guides management.

Diagnostic Approach

History Taking:
-Mechanism of injury: blunt (high-energy impact, deceleration injuries, seatbelt trauma) versus penetrating (stab wounds, gunshot wounds)
-Time since injury
-Associated injuries or comorbidities
-Previous abdominal surgeries or vascular disease
-Vital signs and hemodynamic status of the patient
-Any medications affecting coagulation.
Physical Examination:
-Thorough abdominal examination: inspection for distension or bruising, auscultation for bowel sounds, palpation for tenderness, guarding, rigidity, and masses
-Assessment for pulsatile masses
-Examination of the chest, back, and extremities for associated injuries
-Monitoring vital signs closely for evidence of ongoing hemorrhage or shock.
Investigations:
-FAST (Focused Assessment with Sonography for Trauma) scan: initial screening for intra-abdominal fluid
-CT angiography (CTA) of the abdomen and pelvis: gold standard for diagnosing SMA injury, delineating the extent of injury (occlusion, dissection, pseudoaneurysm, active extravasation), and identifying associated injuries
-Laboratory tests: complete blood count (CBC) for hemoglobin and hematocrit, coagulation profile (PT/INR, PTT), lactate levels (marker of ischemia), type and crossmatch for blood transfusion
-Angiography: can be diagnostic and therapeutic (embolization) for active bleeding or dissection.
Differential Diagnosis:
-Other mesenteric artery injuries (e.g., IMA injury)
-Blunt aortic injury
-Hollow viscus perforation
-Solid organ injury (spleen, liver, kidney)
-Pancreatic contusion or transection
-Bowel obstruction
-Mesenteric venous thrombosis
-Non-traumatic mesenteric ischemia.

Management

Initial Management:
-Immediate resuscitation with intravenous fluids and blood products to stabilize hemodynamic status
-Airway, breathing, circulation (ABC) management
-Prompt surgical exploration is often indicated in hemodynamically unstable patients with suspected SMA injury
-Pain control
-Nasogastric tube decompression.
Medical Management:
-Not primary for acute SMA injury
-Supportive care includes fluid resuscitation, blood product transfusion, and anticoagulation is generally contraindicated in acute active bleeding but may be considered in specific cases of dissection or non-occlusive thrombus without active extravasation, after consultation with vascular surgery.
Surgical Management:
-Exploratory laparotomy is typically indicated
-Goals are to control hemorrhage, assess bowel viability, and revascularize or excise compromised bowel
-Control of proximal and distal SMA (may require mobilizing colon, opening the small bowel mesentery)
-If an intact artery with intimal injury or dissection: endovascular repair (stent graft) or open repair (vein patch angioplasty, graft interposition)
-If complete occlusion/transection with viable bowel: bypass graft (autogenous vein, synthetic graft) or endovascular stenting
-If non-viable bowel: resection of ischemic bowel with primary anastomosis or ostomy
-Ligation of distal SMA branches may be necessary
-Embolectomy may be attempted if thrombus is present
-Damage control surgery may be employed for severe associated injuries or instability.
Supportive Care:
-Intensive care unit (ICU) monitoring post-operatively
-Continued fluid and electrolyte management
-Nutritional support (parenteral or enteral feeding as tolerated)
-Antibiotic prophylaxis
-Monitoring for signs of post-operative ischemia or complications.

Complications

Early Complications: Hemorrhagic shock, bowel infarction, sepsis, anastomotic leak, graft occlusion, retained viable bowel after initial assessment, re-bleeding, compartment syndrome, ARDS, renal failure.
Late Complications: Adhesions and bowel obstruction, incisional hernia, pseudoaneurysm formation at repair site, graft infection, chronic mesenteric ischemia due to compromised collateral flow, short bowel syndrome if extensive resection is required, malabsorption.
Prevention Strategies: Meticulous surgical technique, careful assessment of bowel viability, adequate revascularization, appropriate graft material selection, aggressive hemodynamic resuscitation, early diagnosis, comprehensive intra-operative imaging, prophylactic antibiotics, judicious use of anticoagulation.

Prognosis

Factors Affecting Prognosis: Hemodynamic stability at presentation, extent of SMA injury, time to definitive treatment, degree of bowel ischemia, associated injuries, presence of sepsis, patient comorbidities, surgical expertise.
Outcomes:
-With prompt diagnosis and management, outcomes can be good
-However, delays in treatment significantly worsen prognosis, with high mortality rates associated with bowel infarction and sepsis
-Survival rates are significantly lower in patients requiring extensive bowel resection
-Successful bypass or repair restores perfusion and allows for recovery of bowel function.
Follow Up:
-Regular clinical review to assess for symptoms of ischemia or graft-related complications
-Imaging (CTA or angiography) may be required at intervals to assess graft patency and integrity
-Dietary modification may be necessary if short bowel syndrome develops
-Long-term monitoring for signs of malabsorption.

Key Points

Exam Focus:
-SMA injury is a critical surgical emergency
-CT angiography is the gold standard for diagnosis
-Management hinges on controlling hemorrhage, assessing bowel viability, and revascularization (bypass or endovascular repair) or resection
-Non-viable bowel leads to high mortality
-Differentiate acute from chronic ischemia management.
Clinical Pearls:
-Always suspect SMA injury in high-energy abdominal trauma, especially with unexplained abdominal pain and shock
-Early vascular surgery consultation is paramount
-Meticulous assessment of bowel viability is key
-if in doubt, resect
-Consider collateral supply to the bowel when deciding on management
-Bypass grafts should ideally be autogenous vein when available and feasible.
Common Mistakes:
-Delaying surgical exploration in hemodynamically unstable patients
-Inadequate bowel viability assessment
-Failure to achieve adequate revascularization
-Misinterpreting CTA findings
-Undertreating associated injuries
-Not considering endovascular options for stable injuries.